cdc-coteauxdegaronne
» » Intimal Hyperplasia (Medical Intelligence Unit)
eBook Intimal Hyperplasia (Medical Intelligence Unit) ePub

eBook Intimal Hyperplasia (Medical Intelligence Unit) ePub

by Philip B. Dobrin

  • ISBN: 1879702959
  • Category: Medicine
  • Subcategory: Medicine
  • Author: Philip B. Dobrin
  • Language: English
  • Publisher: R G Landes Co (October 1, 1994)
  • Pages: 355
  • ePub book: 1784 kb
  • Fb2 book: 1428 kb
  • Other: docx txt azw lrf
  • Rating: 4.7
  • Votes: 516

Description

Intimal Hyperplasia book. Intimal Hyperplasia (Medical Intelligence Unit). 1879702959 (ISBN13: 9781879702950).

Intimal Hyperplasia book.

Medical Intelligence Unit. We can notify you when this item is back in stock.

Development of Aneurysms (Medical Intelligence Unit, 17. Richard R. Keen, Philip B. Dobrin.

Development of Aneurysms (Medical Intelligence Unit, 17). Development of Aneurysms (Medical Intelligence Unit, 17). Download (pdf, . 9 Mb) Donate Read. Epub FB2 mobi txt RTF.

Intimal hyperplasia is the thickening of the tunica intima of a blood vessel as a complication of a reconstruction procedure or endarterectomy

Intimal hyperplasia is the thickening of the tunica intima of a blood vessel as a complication of a reconstruction procedure or endarterectomy. Intimal hyperplasia is the universal response of a vessel to injury and is an important reason of late bypass graft failure, particularly in vein and synthetic vascular grafts. Intimal hyperplasia, the obstacle in bypass grafts".

Intimal hyperplasia is not a true disease, but a physiologic healing response to injury to the blood vessel wall Intimal hyperplasia may result from endovascular intervention, vascular surgical procedures, long term vascular catheters and devices, or turbulent flow.

Intimal hyperplasia is not a true disease, but a physiologic healing response to injury to the blood vessel wall. It is the bane of endovascular intervention and vascular surgery. When the endothelium is injured, endothelial cells release inflammatory mediators that trigger platelet aggregation, fibrin deposition and recruitment of leukocytes to the area. Intimal hyperplasia may result from endovascular intervention, vascular surgical procedures, long term vascular catheters and devices, or turbulent flow. It is not infrequently seen after angioplasty, stent insertion, around long term venous catheters, and at surgical vascular and graft anastomoses.

However, normal or "benign" intimal hyperplasia, although microscopically identical to pathology, is a controllable phenotype that rarely compromises blood supply

However, normal or "benign" intimal hyperplasia, although microscopically identical to pathology, is a controllable phenotype that rarely compromises blood supply

Finding books BookSee BookSee - Download books for free.

Finding books BookSee BookSee - Download books for free.

Thrombolytic Therapy Balloon Catheter Vein Graft Intimal Hyperplasia Pull Force. Dobrin PB, Gray J, Schwarcz TH. In vivo models used to study myointimal hyperplasia. These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves. In: Dobrin PB, ed. Intimal Hyperplasia, Austin TX: . Landes C. 1994, pp 75–83. 11. Chidi C, DePalma RG.

DOBRIN, PHILIP . AND ALLEN A, &NICK Autogenous veins undergo intimal hyperplasia and medial thickening when used as arterial bypass grafts

DOBRIN, PHILIP . AND ALLEN A, &NICK. hj%ence of mmlmsmooth muscle on contractile meclzanics and elasticity of arteries, Am, J. Physiol. Autogenous veins undergo intimal hyperplasia and medial thickening when used as arterial bypass grafts. Exposing veins to arterial pressure and flow subjects them to three static deformations, thre. More).

Intimal hyperplasia may be defined as the abnormal migration and proliferation of vascular smooth muscle cells with associated deposition of extracellular connective tissue matrix.

Examining the diagnosis, aetiology and treatment of myointimal hyperplasmia, this volume provides information on the clinical manifestations of myointimal hyperplasia, endovascular ultrasonographic diagnosis and endovascular treatment. It highlights the histologic and electron microscopic features of the process and describes mechanical, cellular and biochemical mechanisms stimulating its development, as well as the pharmacologic methods that may inhibit it.