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eBook Respiratory Hypersensitivity: Its Cholinergic Background  Treatment ePub

eBook Respiratory Hypersensitivity: Its Cholinergic Background Treatment ePub

  • ISBN: 0946270449
  • Subcategory: No category
  • Language: English
  • Publisher: Hyperion Books
  • ePub book: 1872 kb
  • Fb2 book: 1735 kb
  • Other: lit doc rtf lrf
  • Rating: 4.2
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Semantic Scholar extracted view of "Respiratory hypersensitivity : its cholinergic . oceedings{1989RespiratoryH, title {Respiratory hypersensitivity : its cholinergic background and treatment}, author {増市 瀧野 and Yoshitada Takino and 辰郎 瀧野}, year {1989} }.

Semantic Scholar extracted view of "Respiratory hypersensitivity : its cholinergic background and treatment" by 瀧野 増市 et a.

Respiratory Hypersensitivity. Its Cholinergic Background & Treatment. Published December 1989 by Hyperion Books.

118+ million publications.

Inhibition of endogenous acetylcholine degradation through cholinesterase inhibitors represents a milestone in symptomatic treatment of cognitive symptoms in mild to moderate stages of Alzheimer's disease. Cholinesterase inhibitors are also under investigation for treating cognitive dysfunction of cerebrovascular origin, but to date they do not have specific indication for vascular dementia or vascular cognitive impairment.

Respiratory hypersensitivity its cholinergic background and treatment. Respiratory hypersensitivity its cholinergic background and treatment.

Cholinergic agents are compounds which mimic the action of acetylcholine and/or butyrylcholine

Cholinergic agents are compounds which mimic the action of acetylcholine and/or butyrylcholine. In general, the word "choline" describes the various quaternary ammonium salts containing the N,N,monium cation. Found in most animal tissues, choline is a primary component of the neurotransmitter acetylcholine and functions with inositol as a basic constituent of lecithin. Choline also prevents fat deposits in the liver and facilitates the movement of fats into cells.

It is concluded that chronic opiate treatment can induce supersensitivity of the cholinergic systems subserving control of respiration. Message Subject (Your Name) has forwarded a page to you from Journal of Pharmacology and Experimental Therapeutics. Message Body (Your Name) thought you would be interested in this article in Journal of Pharmacology and Experimental Therapeutics. Flag as Inappropriate. In type 1 hypersensitivity, an antigen is presented to CD4+ Th2 cells specific to the antigen that stimulate B-cell production of IgE antibodies also specific to the antigen. The difference between a normal infectious immune response and a type 1 hypersensitivity response is that in type 1 hypersensitivity the antibody is IgE instead of IgA, IgG, or IgM. During sensitisation, the IgE antibodies bind to Fcε receptors on the surface of tissue mast cells and blood basophils. Mast cells and basophils coated by IgE antibodies are "sensitised.